Is there a Hispanic Health Paradox in Sensitivity to Air Pollution? Hospital Admissions for Asthma, Chronic Obstructive Pulmonary Disease and Congestive Heart Failure Associated with NO(2) and PM(2.5) in El Paso, TX, 2005-2010

是否存在西班牙裔人群对空气污染敏感性的健康悖论?2005-2010年德克萨斯州埃尔帕索市与NO(2)和PM(2.5)相关的哮喘、慢性阻塞性肺病和充血性心力衰竭住院病例

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Abstract

STUDY OBJECTIVE: Linkages between pollution and morbidity have been observed in numerous studies. But race/ethnicity has been underemphasized as a modifier of that association, and few studies have tested for a Hispanic Health Paradox in sensitivity to air pollution. METHODS: Daily asthma, chronic obstructive pulmonary disease (COPD) and congestive heart failure (CHF) hospital admissions in El Paso, Texas were studied in age groups and insurance groups. Daily PM(2.5) and NO(2) were calculated from pollution monitors and all models adjusted for apparent temperature and wind speed. Conditional logistic regression for the case-crossover design was used for a between-group comparison and for a within-group comparison for Hispanics. RESULTS: Hispanics were at lower risk than non-Hispanic whites and non-Hispanics of other races for NO(2)-associated admissions, but at greater risk for PM(2.5)-associated admissions. While Hispanics were generally protected with regards to NO(2), Hispanic children (vs. elderly) faced increased risk for asthma and uninsured Hispanics (vs. Private) faced increased risk for COPD admissions. While Hispanics were at increased risk of PM(2.5)-associated admissions, certain characteristics heightened their risks: being a Hispanic child (vs. Elderly) for asthma; being a Hispanic with Medicare (vs. Private) for asthma; and being a Hispanic with private insurance (vs. all other insurance types) for CHF. The main effect of pollution on admissions was more significant for asthma and CHF than for COPD, which had the fewest cases. CONCLUSIONS: There was heterogeneity in sensitivity to air pollution based on social characteristics and moderate evidence for a Hispanic Health Paradox in sensitivity to NO(2).

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