Neurobiology of Pain

Enhanced trafficking of an inherited erythromelalgia Na(V)1.7 mutant channel at a physiological temperature.

在生理温度下，遗传性红斑肢痛症 Na(V)1.7 突变通道的转运增强

Targeting Na(v)1.7 and Na(v)1.8 with a PIKfyve inhibitor to reverse inflammatory and neuropathic pain.

使用 PIKfyve 抑制剂靶向 Na(v)1.7 和 Na(v)1.8 来逆转炎症性和神经性疼痛

R and S enantiomers of CBD3063, a Ca(V)2.2 N-type calcium channel modulator, alleviate capsaicin-induced inflammatory pain.

CBD3063 的 R 和 S 对映体（一种 Ca(V)2.2Â N 型钙通道调节剂）可缓解辣椒素引起的炎症疼痛

ASIC1a-associated mechanical hypersensitivity in the GlaKO Fabry disease mouse model.

GlaKO Fabry 病小鼠模型中 ASIC1a 相关的机械性过敏

Humanized Na(V)1.8 rats overcome cross-species potency shifts in developing novel Na(V)1.8 inhibitors.

人源化 Na(V)1.8 大鼠克服了开发新型 Na(V)1.8 抑制剂时跨物种效力变化的问题

NGF stimulation alters the transcriptome and surface TrkB expression in axons of dorsal root ganglion neurons.

NGF刺激会改变背根神经节神经元轴突的转录组和表面TrkB表达

Sodium channels Na(v)1.7, Na(v)1.8 and pain; two distinct mechanisms for Na(v)1.7 null analgesia.

Neurokinin1 - cholinergic receptor mechanisms in the medial Septum-Dorsal hippocampus axis mediates experimental neuropathic pain.

神经激肽1 - 内侧隔核-背侧海马轴中的胆碱能受体机制介导实验性神经性疼痛

Keratinocyte-derived extracellular vesicles in painful diabetic neuropathy

糖尿病疼痛性神经病变中的角质形成细胞衍生的细胞外囊泡

B-cell and plasma cell activation in a mouse model of chronic muscle pain

慢性肌肉疼痛小鼠模型中的 B 细胞和浆细胞活化