Journal of Pineal Research

期刊全称

期刊缩写

J. Pineal Res.

涉及主题

医学生物内科学内分泌学心理学神经科学褪黑素化学生物化学遗传学昼夜节律物理有机化学细胞生物学松果体受体核磁共振酶基因病理

期刊介绍

Journal of Pineal Research is a forum for the original scientific results of basic, applied, and clinical research involving any aspect of the pineal gland or its hormonal products in all vertebrate species. The journal also fully considers the biological functions and activities of melatonin in non-vertebrate species, plants and microorganisms. Essential criteria for publication are scientific importance, novelty, and timeliness, as well as clarity of presentation. Experimental data that are contrary to currently accepted thinking or divergent from actual dogma will be considered, provided the findings are based on solid, new experimental observations. All submitted papers must be complete in themselves. Case reports will be considered if the findings contribute significantly to an understanding of some aspect of the pineal gland and melatonin research. It is the expressed aim of the journal to serve pineal gland and melatonin researchers in all disciplines.

期刊ISSN

print: 0742-3098

历年影响因子

2024年	2023年	2022年	2021年	2020年	2019年	2018年	2017年
8.3	10.3	12.081	13.007	14.528	15.221	11.613	10.391

历年发表/被引量

年份	2025	2024	2023	2022	2021	2020	2019	2018	2017	2016	2015	2014	2013	2012
发表量	44	117	77	74	81	92	91	97	89	87	109	95	86	107
被引量	4955	12391	13834	14821	14796	13881	11895	12072	10409	8966	7875	8674	7568	6627

h-Index

113

自引率

4.80%

涉及的研究领域

医学-内分泌学与代谢

中科院2025年分区

大类	小类	TOP期刊	综述期刊
1区	1区内分泌学与代谢	是	否
	1区神经科学
	1区生理学

WOS期刊分区

JCR学科分类

JCR分区学科名称	收录数据库	JCR分区	分区排名
ENDOCRINOLOGY & METABOLISM	SCIE	Q1	24/191
NEUROSCIENCES	SCIE	Q1	33/314
PHYSIOLOGY	SCIE	Q1	4/87

JCI学科分类

JCI分区学科名称	收录数据库	JCI分区	分区排名
ENDOCRINOLOGY & METABOLISM	SCIE	Q1	10/191
NEUROSCIENCES	SCIE	Q1	18/314
PHYSIOLOGY	SCIE	Q1	3/87

期刊主页

http://onlinelibrary.wiley.com/journal/10.1111/(ISSN)1600-079X

投稿网址

https://mc.manuscriptcentral.com/jpi

出版商

Wiley-Blackwell Publishing Ltd

出版国家（地区）

UNITED STATES

出版语言

English

出版周期

Bimonthly

每年出版文章数

Gold OA文章占比

22.56%

原创研究文献占比
(排除综述)

90.74%

SCI收录类型

Science Citation Index Expanded (SCIE)

Scopus (CiteScore)

PubMed链接

http://www.ncbi.nlm.nih.gov/nlmcatalog?term=0742-3098%5BISSN%5D

平均审稿周期

平均1.0个月

平均录用比例

较难

Journal of Pineal Research

JCR学科分类

JCI学科分类

期刊相关文献 (158)

Sex-Specific Methylomic and Transcriptomic Responses of the Avian Pineal Gland to Unpredictable Illumination Patterns.

Melatonin Repairs the Lipidome of Human Hepatocytes Exposed to Cd and Free Fatty Acid-Induced Lipotoxicity.

褪黑素可修复暴露于镉和游离脂肪酸诱导的脂毒性的人类肝细胞的脂质组

Melatonin Deficits Result in Pathologic Metabolic Reprogramming in Differentiated Neurons.

褪黑激素缺乏会导致分化神经元发生病理性代谢重编程

Orphan GPR50 Restrains Neurite Outgrowth and Cell Migration by Activating the G(12/13) Protein-RhoA Pathway in Neural Progenitor Cells and Tanycytes.

孤儿 GPR50 通过激活神经祖细胞和室管膜细胞中的 G(12/13) 蛋白-RhoA 通路来抑制神经突生长和细胞迁移

Melatonin Increased Autophagy Level to Facilitate Osteogenesis of Inflamed PDLSCs Through TMEM110 Signaling Pathways.

褪黑素通过TMEM110信号通路增加自噬水平，促进炎症性牙周膜干细胞的成骨作用

Modeling Genetic Risk of Î²-Cell Dysfunction in Human Induced Pluripotent Stem Cells From Patients Carrying the MTNR1B Risk Variant.

对携带 MTNR1B 风险变异的患者的人诱导多能干细胞中 β 细胞功能障碍的遗传风险进行建模

Melatonin Induces PERK-ATF4 Unfolded Protein Response and Apoptosis in Human Choriocarcinoma Cells.

褪黑素诱导人绒毛膜癌细胞中 PERK-ATF4 未折叠蛋白反应和细胞凋亡

Multi-Omics Analysis Reveals That AhNHL Contributes to Melatonin-Mediated Cadmium Tolerance in Peanut Plants.

多组学分析揭示 AhNHL 有助于花生植株褪黑素介导的镉耐受性

Light-Dependent Circadian Rhythm Governs O-GlcNAc Cycling to Influence Cognitive Function in Adult Zebrafish

光依赖性昼夜节律控制 O-GlcNAc 循环以影响成年斑马鱼的认知功能

KARS Mutations Impair Brain Myelination by Inducing Oligodendrocyte Deficiency: One Potential Mechanism and Improvement by Melatonin

KARS 突变通过诱导少突胶质细胞缺陷而损害脑髓鞘形成：一种潜在机制及褪黑激素的改善作用