Lubiprostone in chronic kidney disease: Insights into mitochondrial function and polyamines from a randomized phase 2 clinical trial.

鲁比前列酮治疗慢性肾脏病:一项随机 2 期临床试验揭示线粒体功能和多胺的见解

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作者:Watanabe Shun, Nakayama Masaaki, Yokoo Takashi, Sanada Satoru, Ubara Yoshifumi, Komatsuda Atsushi, Asanuma Katsuhiko, Suzuki Yusuke, Konta Tsuneo, Kazama Junichiro J, Suzuki Takehiro, Fukuda Shinji, Soga Tomoyoshi, Yamada Takuji, Mizutani Sayaka, Matsumoto Mitsuharu, Naito Yuji, Taguchi Kensei, Fukami Kei, Kashiwagi Hitomi, Kikuchi Koichi, Suzuki Chitose, Tokuno Hidetaka, Urasato Marina, Kujirai Ryota, Matsumoto Yotaro, Akiyama Yasutoshi, Tomioka Yoshihisa, Itai Shun, Tongu Yoshiyasu, Mishima Eikan, Kawabe Chiharu, Kasahara Tomoko, Ogata Yoshiaki, Toyohara Takafumi, Sato Takeya, Tanaka Tetsuhiro, Abe Takaaki
Chronic kidney disease (CKD) is a life-threatening condition, and constipation is a progressive risk factor. We evaluated changes in uremic toxins, renal function, and the safety of lubiprostone, a selective chloride channel activator, in patients with CKD. In this phase 2, randomized, double-blind, placebo-controlled trial across nine centers in Japan, 150 patients with stage IIIb-IV CKD received lubiprostone (8 or 16 micrograms) or placebo for 24 weeks. The primary end point was change in indoxyl sulfate levels. Secondary end points included other uremic toxins and renal function markers. Lubiprostone did not alter uremic toxin levels but improved or preserved estimated glomerular filtration rate and its slope in the 16-microgram group. Mild-to-moderate gastrointestinal events occurred in the placebo and 16-microgram groups. Multiomics analysis revealed that lubiprostone modulated the gut microbial agmatine pathway and increased spermidine levels, thereby improving renal mitochondrial function. Lubiprostone is a previously unknown and safe therapeutic option to mitigate renal decline in CKD.

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