The aim of the study was to investigate the therapeutic potential of exosomes secreted by endothelial cells (EC-exos) on steroid-induced osteonecrosis of femoral head (SNFH). First, we successfully obtained EC-exos through differential centrifugation. Then, the effects of EC-exos on mouse embryo osteoblast precursor (MC3T3-E1) cells under high concentration of dexamethasone (Dex) were analysed in vitro, which included cell migration, viability, and apoptosis. In vivo, a SNFH rat model was successfully established and treated with EC-exos. Micro-computed tomography (micro-CT) and haematoxylin and eosin (H&E) were used to observe femoral trabeculae. Our in vitro results showed that EC-exos improved cell viability and migration of osteoblasts and reduced the apoptotic effect of high concentration of Dex on osteoblasts in vitro. Phosphoinositide 3-kinase (PI3K)/Akt/Bcl-2 signalling pathway was activated in MC3T3-E1 cells under the response to EC-exos. In vivo, increased bone volume per tissue volume (BV/TV) (p=0.031), trabecular thickness (Tb.Th) (p=0.020), and decreased separation (Tb.Sp) (p=0.040) were observed in SNFH rats treated with EC-exos. H&E staining revealed fewer empty lacunae and pyknotic osteocytes in trabeculae. The expression of Bcl-2 and Akt in EC-exos group was significantly increased in trabeculae tissue. Overall, our finding indicated that EC-exos could attenuate SNFH by inhibiting osteoblast apoptosis via the PI3K/Akt/Bcl-2 pathway.
Endothelial Cell-Derived Exosomes Inhibit Osteoblast Apoptosis and Steroid-Induced Necrosis of Femoral Head Progression by Activating the PI3K/Akt/Bcl-2 Pathway.
内皮细胞衍生的外泌体通过激活 PI3K/Akt/Bcl-2 通路抑制成骨细胞凋亡和类固醇诱导的股骨头坏死进展
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作者:Sun Jie, Yao Chen, Luo Wanxin, Ge Xingyu, Zheng Wenjie, Sun Chi, Zhang Yafeng
| 期刊: | Journal of Tissue Engineering and Regenerative Medicine | 影响因子: | 2.600 |
| 时间: | 2024 | 起止号: | 2024 May 10; 2024:3870988 |
| doi: | 10.1155/2024/3870988 | 研究方向: | 细胞生物学 |
| 信号通路: | Apoptosis、PI3K/Akt | ||
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