Targeting transcription factors through an IMiD independent zinc finger domain.

通过不依赖于IMiD的锌指结构域靶向转录因子

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作者:Liu Bee Hui, Liu Miao, Radhakrishnan Sridhar, Dai Meng-Yuan, Jaladanki Chaitanya Kumar, Gao Chong, Tang Jing Ping, Kumari Kalpana, Go Mei Lin, Vu Kim Anh L, Kwon Junsu, Seo Hyuk-Soo, Song Kijun, Tian Xi, Feng Li, Tan Justin L, Melkonian Arek V, Liu Zhaoji, Wulf Gerburg, Arthanari Haribabu, Qi Jun, Dhe-Paganon Sirano, Clohessy John G, Choong Yeu Khai, Sivaraman J, Fan Hao, Tenen Daniel G, Chai Li
Immunomodulatory imide drugs (IMiDs) degrade specific C2H2 zinc finger degrons in transcription factors, making them effective against certain cancers. SALL4, a cancer driver, contains seven C2H2 zinc fingers in three clusters, including an IMiD degron in zinc finger cluster one (ZFC1). Surprisingly, IMiDs do not inhibit the growth of SALL4-expressing cancer cells. To overcome this limit, we focused on a non-IMiD domain, SALL4 zinc finger cluster four (ZFC4). By combining ZFC4-DNA crystal structure and an in silico docking algorithm, in conjunction with cell viability assays, we screened several chemical libraries against a potentially druggable binding pocket, leading to the discovery of SH6, a compound that selectively targets SALL4-expressing cancer cells. Mechanistic studies revealed that SH6 degrades SALL4 protein through the CUL4A/CRBN pathway, while deletion of ZFC4 abolished this activity. Moreover, SH6 treatment led to a significant 87% tumor growth inhibition of SALL4+ patient-derived xenografts and demonstrated good bioavailability in pharmacokinetic studies. In summary, these studies represent a new approach for IMiD independent drug discovery targeting C2H2 transcription factors such as SALL4 in cancer.

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