Inflammation plays a central role in various pathological conditions, necessitating the search for safer and more effective anti-inflammatory agents. This study investigates the anti-inflammatory activity of caulerpin, a bisindolic alkaloid isolated from Caulerpa racemosa. In vitro assays demonstrated that caulerpin significantly reduced nitric oxide, TNF-α, IL-6, and IL-12 levels in macrophages stimulated with LPS + IFN-γ, without affecting cell viability. In silico toxicity predictions using Protox 3.0 reinforce a favorable safety profile of caulerpin. Molecular docking and molecular dynamics simulations revealed its high-affinity binding to the glucocorticoid receptor ligand-binding domain (GR-LBD), suggesting a mechanism of action similar to dexamethasone. The involvement of the glucocorticoid receptor was confirmed by the partial reversal of caulerpin's effects upon RU486 treatment. In vivo, caulerpin exhibited a favorable safety profile, with no signs of acute toxicity at an oral dose of 100 mg/kg. Moreover, in a mouse model of endotoxic shock, caulerpin administration significantly improved survival rates in a dose-dependent manner, providing complete protection at 4 mg/kg. These findings highlight caulerpin as a promising candidate for the development of novel anti-inflammatory therapies. Further studies are warranted to explore its pharmacokinetics, optimize its structure, and evaluate its efficacy in chronic inflammatory diseases.
The Alkaloid Caulerpin Exhibits Potent and Selective Anti-Inflammatory Activity Through Interaction with the Glucocorticoid Receptor.
生物碱考勒平通过与糖皮质激素受体相互作用,表现出强效且选择性的抗炎活性
阅读:9
作者:Dos Santos Jônatas Sousa Pires, Silva Dahara Keyse Carvalho, da Silva Oliveira Vanessa, Junior Sergio Santos Silva, Dos Santos Rodrigues Edivaldo, de Souza Claudia Valeria Campos, Martinez Sabrina Teixeira, Santos-Filho Osvaldo Andrade, Meira Cássio Santana, Soares Milena Botelho Pereira
| 期刊: | Marine Drugs | 影响因子: | 5.400 |
| 时间: | 2025 | 起止号: | 2025 May 29; 23(6):232 |
| doi: | 10.3390/md23060232 | 研究方向: | 炎症/感染 |
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