Reports focusing on the immunological microenvironment of peripheral T-cell lymphomas (PTCL) are rare. Here we studied the reciprocal contribution of regulatory (Treg) and interleukin-17-producing (Th17) T-cells to the composition of the lymphoma-associated microenvironment of angioimmunoblastic T-cell lymphoma (AITL) and PTCL not otherwise specified on tissue microarrays from 30 PTCLs not otherwise specified and 37 AITLs. We found that Th17 but not Treg cells were differently represented in the two lymphomas and correlated with the amount of mast cells (MCs) and granulocytes, which preferentially occurred in the cellular milieu of AITL cases. We observed that MCs directly synthesized interleukin-6 and thus contribute to the establishment of a pro-inflammatory, Th17 permissive environment in AITL. We further hypothesized that the AITL clone itself could be responsible for the preferential accumulation of MCs at sites of infiltration through the synthesis of CXCL-13 and its interaction with the CXCR3 and CXCR5 receptors expressed on MCs. Consistent with this hypothesis, we observed MCs efficiently migrating in response to CXCL-13. On these bases, we conclude that MCs have a role in molding the immunological microenvironment of AITL toward the maintenance of pro-inflammatory conditions prone to Th17 generation and autoimmunity.
Mast cells and Th17 cells contribute to the lymphoma-associated pro-inflammatory microenvironment of angioimmunoblastic T-cell lymphoma.
肥大细胞和 Th17 细胞促进血管免疫母细胞性 T 细胞淋巴瘤的淋巴瘤相关促炎微环境
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作者:Tripodo Claudio, Gri Giorgia, Piccaluga Pier Paolo, Frossi Barbara, Guarnotta Carla, Piconese Silvia, Franco Giovanni, Vetri Valeria, Pucillo Carlo Ennio, Florena Ada Maria, Colombo Mario Paolo, Pileri Stefano Aldo
| 期刊: | American Journal of Pathology | 影响因子: | 3.600 |
| 时间: | 2010 | 起止号: | 2010 Aug;177(2):792-802 |
| doi: | 10.2353/ajpath.2010.091286 | 研究方向: | 细胞生物学 |
| 疾病类型: | 淋巴瘤 | ||
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