Non-alcoholic fatty liver disease (NAFLD) is the most common chronic liver disease, projected to affect 55% globally by 2040. Up to one-third of NAFLD patients develop non-alcoholic steatohepatitis (NASH), with 40% progressing to fibrosis. However, there are currently few reliable tools to predict disease progression. Impaired mitochondrial dynamics, characterized by dysregulated fission, fusion, and mitophagy, have emerged as key events in NAFLD pathophysiology, contributing to hepatocyte death and inflammation. This study explored the transition from steatosis to NASH through transcriptomic analyses, including data from patients with steatosis and those with NASH at different fibrosis stages. By identifying a transcriptomic signature associated with disease progression, the study revealed increased expression of genes involved in mitochondrial dynamics in NASH compared to steatosis and during NASH-related fibrosis. Histological analyses highlighted the central role of Dynamin-related protein 1 (Drp1), a dynamin GTPase essential for mitochondrial fission and mitophagy. In human liver biopsies, Drp1 expression progressively increased from NAFLD to NASH and NASH-related fibrosis and cirrhosis, predominantly in Kupffer cells. These finding suggest Drp1 is a potential driver of the transition to more severe liver damage, making it a promising biomarker for NASH development and progression and a potential therapeutic target in metabolic disorders.
Analysis of the Mitochondrial Dynamics in NAFLD: Drp1 as a Marker of Inflammation and Fibrosis.
NAFLD 中线粒体动力学的分析:Drp1 作为炎症和纤维化的标志物
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作者:Padelli Maël, Hamelin Jocelyne, Desterke Christophe, Sebagh Mylène, Saffroy Raphael, Sanchez Claudio Garcia, Coilly Audrey, Duclos-Vallée Jean-Charles, Samuel Didier, Lemoine Antoinette
| 期刊: | International Journal of Molecular Sciences | 影响因子: | 4.900 |
| 时间: | 2025 | 起止号: | 2025 Jul 30; 26(15):7373 |
| doi: | 10.3390/ijms26157373 | 研究方向: | 免疫/内分泌 |
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