A shared inflammatory signature across severe malaria syndromes manifested by transcriptomic, proteomic and metabolomic analyses.

转录组学、蛋白质组学和代谢组学分析表明,重症疟疾综合征具有共同的炎症特征

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作者:Sobota Rafal S, Stucke Emily M, Coulibaly Drissa, Lawton Jonathan G, Cummings Bryan E, Sebastian Savy, Dara Antoine, Munro James B, Ouattara Amed, Kone Abdoulaye K, Kane Bourama, Traoré Karim, Guindo Bouréima, Tangara Bourama M, Niangaly Amadou, Ventimiglia Noah T, Daou Modibo, Diarra Issa, Tolo Youssouf, Sissoko Mody, Maiga Fayçal, Diawara Aichatou, Traore Amidou, Thera Ali, Laurens Matthew B, Lyke Kirsten E, Kouriba Bourema, Doumbo Ogobara K, Plowe Christopher V, Goodlett David R, Silva Joana C, Thera Mahamadou A, Travassos Mark A
Factors governing the clinical trajectory of Plasmodium falciparum infection remain an important area of investigation. Here we present transcriptomic, proteomic and metabolomic analyses comparing clinical subtypes of severe Plasmodium falciparum malaria to matched controls with uncomplicated disease in 79 children from Mali. MMP8, IL1R2, and ARG1 transcription is higher across cerebral malaria, severe malarial anemia, and concurrent cerebral malaria and severe malarial anemia, indicating a shared inflammatory signature. Tissue inhibitor of metalloproteinases 1 is the most upregulated protein in cerebral malaria, which along with elevated MMP8 and MMP9 transcription, underscores the importance of the metalloproteinase pathway in central nervous system pathophysiology. L-arginine metabolites are decreased in cerebral malaria, which coupled with increased ARG1 transcription suggests a putative mechanism impairing cerebral vasodilation. Using multi-omics approaches, we thus describe the inflammatory cascade in severe malaria syndromes, and identify potential therapeutic targets and biological markers.

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