Usher syndrome (USH) is the most common cause of deafblindness. USH is autosomal recessively inherited and characterized by rod-cone dystrophy or retinitis pigmentosa (RP), often accompanied by sensorineural hearing loss. Variants in >15 genes have been identified as causative for clinically and genetically distinct subtypes. Among the ultra-rare and recently discovered genes is ARSG, coding for the lysosomal sulfatase Arylsulfatase G. This subtype was assigned as "USH IV" with a late onset of RP and usually late-onset progressive SNHL without vestibular involvement. Here, we describe nine new subjects and the clinical description of four cases with the USH IV phenotype bearing seven novel and two known pathogenic variants. Functional experiments indicated the complete loss of sulfatase enzymatic activity upon ectopic expression of mutated ARSG cDNA. Interestingly, we identified a homozygous missense variant, p.(Arg99His), previously described in dogs with neuronal ceroid lipofuscinosis. Our study expands the genetic landscape of ARSG-USH IV and the number of known subjects by more than 30%. These findings highlight that USH IV likely has been underdiagnosed and emphasize the need to test molecularly unresolved subjects with deafblindness syndrome. Finally, testing of ARSG should be considered for the genetic work-up of apparent isolated inherited retinal diseases.
Expanding the genetic landscape of Usher syndrome type IV caused by pathogenic ARSG variants.
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作者:Bauwens Miriam, De Man Vincent, Audo Isabelle, Balikova Irina, Zein Wadih M, Smirnov Vasily, Held Sebastian, Vermeer Sascha, Loos Elke, Jacob Julie, Casteels Ingele, Désir Julie, Depasse Fanny, Van de Sompele Stijn, Van Heetvelde Mattias, De Bruyne Marieke, Andrieu Camille, Condroyer Christel, Antonio Aline, Hufnagel Robert, Carvalho Ana LuÃsa, Marques João Pedro, Zeitz Christina, De Baere Elfride, Damme Markus
| 期刊: | Clinical Genetics | 影响因子: | 2.300 |
| 时间: | 2025 | 起止号: | 2025 Jan;107(1):44-55 |
| doi: | 10.1111/cge.14614 | ||
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