It is well known that dopamine imbalances are associated with many psychiatric disorders and that the dopaminergic receptor Dâ is the main target of antipsychotics. Recently it was shown that levels of two proteins implicated in dopaminergic signaling, Neuronal calcium sensor-1 (NCS-1) and DARPP-32, are altered in the prefrontal cortex (PFC) of both schizophrenic and bipolar disorder patients. NCS-1, which inhibits Dâ internalization, is upregulated in the PFC of both patients. DARPP-32, which is a downstream effector of dopamine signaling, integrates the pathways of several neurotransmitters and is downregulated in the PFC of both patients. Here, we used PC12 cells stably overexpressing NCS-1 (PC12-NCS-1 cells) to address the function of this protein in DARPP-32 signaling pathway in vitro. PC12-NCS-1 cells displayed downregulation of the cAMP/PKA pathway, with decreased levels of cAMP and phosphorylation of CREB at Ser133. We also observed decreased levels of total and phosphorylated DARPP-32 at Thr34. However, these cells did not show alterations in the levels of Dâ and phosphorylation of DARPP-32 at Thr75. These results indicate that NCS-1 modulates PKA/cAMP signaling pathway. Identification of the cellular mechanisms linking NCS-1 and DARPP-32 may help in the understanding the signaling machinery with potential to be turned into targets for the treatment of schizophrenia and other debilitating psychiatric disorders.
Downregulation of the cAMP/PKA pathway in PC12 cells overexpressing NCS-1.
在过表达 NCS-1 的 PC12 细胞中,cAMP/PKA 通路下调
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作者:Souza Bruno R, Torres Karen C L, Miranda Débora M, Motta Bernardo S, Caetano Fernando S, Rosa Daniela V F, Souza Renan P, Giovani Antônio Jr, Carneiro Daniel S, Guimarães Melissa M, Martins-Silva Cristina, Reis Helton J, Gomez Marcus V, Jeromin Andreas, Romano-Silva Marco A
| 期刊: | Cellular and Molecular Neurobiology | 影响因子: | 4.800 |
| 时间: | 2011 | 起止号: | 2011 Jan;31(1):135-43 |
| doi: | 10.1007/s10571-010-9562-4 | 研究方向: | 细胞生物学 |
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