Kinase inhibitors constitute an important new class of cancer drugs, whose selective efficacy is largely determined by underlying tumor cell genetics. We established a high-throughput platform to profile 500 cell lines derived from diverse epithelial cancers for sensitivity to 14 kinase inhibitors. Most inhibitors were ineffective against unselected cell lines but exhibited dramatic cell killing of small nonoverlapping subsets. Cells with exquisite sensitivity to EGFR, HER2, MET, or BRAF kinase inhibitors were marked by activating mutations or amplification of the drug target. Although most cell lines recapitulated known tumor-associated genotypes, the screen revealed low-frequency drug-sensitizing genotypes in tumor types not previously associated with drug susceptibility. Furthermore, comparing drugs thought to target the same kinase revealed striking differences, predictive of clinical efficacy. Genetically defined cancer subsets, irrespective of tissue type, predict response to kinase inhibitors, and provide an important preclinical model to guide early clinical applications of novel targeted inhibitors.
Identification of genotype-correlated sensitivity to selective kinase inhibitors by using high-throughput tumor cell line profiling.
利用高通量肿瘤细胞系分析鉴定与基因型相关的对选择性激酶抑制剂的敏感性
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作者:McDermott Ultan, Sharma Sreenath V, Dowell Lori, Greninger Patricia, Montagut Clara, Lamb Jennifer, Archibald Heidi, Raudales Raul, Tam Angela, Lee Diana, Rothenberg S Michael, Supko Jeffrey G, Sordella Raffaella, Ulkus Lindsey E, Iafrate A John, Maheswaran Shyamala, Njauw Ching Ni, Tsao Hensin, Drew Lisa, Hanke Jeff H, Ma Xiao-Jun, Erlander Mark G, Gray Nathanael S, Haber Daniel A, Settleman Jeffrey
| 期刊: | Proceedings of the National Academy of Sciences of the United States of America | 影响因子: | 9.100 |
| 时间: | 2007 | 起止号: | 2007 Dec 11; 104(50):19936-41 |
| doi: | 10.1073/pnas.0707498104 | 研究方向: | 肿瘤 |
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