Metabolic dysfunction-associated steatotic liver disease (MASLD) is a progressive disease and comprises different stages of liver damage; it is significantly associated with obese and overweight patients. Untreated MASLD can progress to life-threatening end-stage conditions, such as cirrhosis and liver cancer. N-Linked glycosylation is one of the most common post-translational modifications in the cell surface and secreted proteins. N-Linked glycan alterations have been established to be signatures of liver diseases. However, the N-linked glycan changes during the progression of MASLD to liver cancer are still unknown. Here, we induced different stages of MASLD in mice and liver-cancer-related phenotypes and elucidated the N-glycome profile during the progression of MASLD by quantitative and qualitative profiling in situ using matrix-assisted laser desorption ionization (MALDI) imaging mass spectrometry (IMS). Importantly, we identified specific N-glycan structures including fucosylated and highly branched N-linked glycans at very early stages of liver injury (steatosis), which in humans are associated with cancer development, establishing the importance of these modifications with disease progression. Finally, we report that N-linked glycan alterations can be observed in our models by MALDI-IMS before liver injury is identified by histological analysis. Overall, we propose these findings as promising biomarkers for the early diagnosis of liver injury in MASLD.
Spatial Omics Reveals that Cancer-Associated Glycan Changes Occur Early in Liver Disease Development in a Western Diet Mouse Model of MASLD.
空间组学揭示,在西方饮食诱导的 MASLD 小鼠模型中,与癌症相关的聚糖变化在肝病发展早期发生
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作者:Ochoa-Rios Shaaron, Grauzam Stéphane Elie, Gregory Rebecca, Angel Peggi M, Drake Richard R, Helke Kristi L, Mehta Anand S
| 期刊: | Journal of Proteome Research | 影响因子: | 3.600 |
| 时间: | 2024 | 起止号: | 2024 Feb 2; 23(2):786-796 |
| doi: | 10.1021/acs.jproteome.3c00672 | 种属: | Mouse |
| 研究方向: | 肿瘤 | ||
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