High expression of interleukin-18 receptor alpha correlates with severe respiratory viral disease and defines T cells with reduced cytotoxic signatures

白细胞介素-18受体α的高表达与严重的呼吸道病毒疾病相关,并定义了细胞毒性特征降低的T细胞。

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作者:Aira F Cabug ,Jeremy Chase Crawford ,Hayley A McQuilten ,Isabelle J H Foo ,Lilith F Allen ,Deborah Gebregzabher ,Robert C Mettelman ,Tanya Novak ,Janet Chou ,Louise C Rowntree ,Ruth R Hagen ,Abby J Thomson ,Genevieve E Martin ,Brad Gilbertson ,Michael Nt Souter ,Fiona James ,Emma Goodall ,Simone Rizzetto ,Tim Flerlage ,Xiaoxiao Jia ,Lee-Ann Van de Velde ,So Young Chang ,Fabio Luciani ,Ryan S Thwaites ,Jason A Trubiano ,Tom C Kotsimbos ,Allen C Cheng ,Adrienne G Randolph ,Paul G Thomas ,Jianqing Xu ,Zhongfang Wang ,Thi H O Nguyen # ,Brendon Y Chua # ,Lukasz Kedzierski # ,Katherine Kedzierska #

Abstract

Hyperactivated immunity underpins severe outcomes of respiratory viral infections, yet specific immune perturbations are ill-defined. Our recent findings identified OLAH (oleoyl-ACP-hydrolase) as a driver of life-threatening viral diseases. In the same patient cohorts, we now identify the gene encoding IL-18Rα chain (IL18R1), as being highly expressed in life-threatening influenza, COVID-19, RSV and multisystem inflammatory syndrome in children (MIS-C) and demonstrate markedly elevated surface protein IL-18Rα expression on CD8 T cells in these infections. Using a mouse model of severe influenza, we further show that high IL-18Rα expression on effector T cells is associated with increased disease severity. We find that IL-18Rα expression on CD8 T cells is inversely associated with cytotoxicity-related genes, including granzyme A, granzyme B, perforin, Eomes, and KLRG-1. Our study demonstrates that IL-18Rα is associated with severe and fatal respiratory disease outcomes and proposes the use of IL-18Rα as a potential biomarker for severe respiratory viral disease.

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