Anesthesia blocks the cardiovascular response to baroreceptor unloading in rats

麻醉剂会阻断大鼠对压力感受器卸载的心血管反应。

阅读:2

Abstract

Baroreflex responsiveness and orthostatic stability in humans can be assessed by a variety of approaches, including exposure to graded levels of lower body negative pressure (LBNP). However, such approaches have limited applicability in animal studies owing to the need to anesthetize or sedate the animal. We recently reported a novel approach for the assessment of baroreceptor responsiveness in the awake rat using LBNP and presented preliminary findings that 3% isoflurane anesthesia completely blocked the normally robust baroreflex. In the present study, we sought to extend these findings by studying the effects of several common anesthetics on LBNP responsiveness. Blood pressure (BP) and heart rate (HR) responses to progressive levels of LBNP were first made in awake rats (male and female), followed by measurements under various anesthetics regimens: 1) pentobarbital; 2) ketamine plus xylazine; 3) isoflurane at 3%, 2%, and 1.5%; 4) urethane delivered as an intraperitoneal bolus, slow intraperitoneal infusion, and slow intravenous infusion. As previously reported, BP in awake rats was well maintained up to -15 mmHg LBNP, accompanied by a robust baroreflex tachycardia. Despite varying effects on steady-state BP and HR, all of the anesthetics tested severely or completely blocked the ability to maintain BP during LBNP and completely blocked reflex tachycardia. BP, but not reflex tachycardia, during LBNP was partially preserved only in those rats treated with intravenous urethane. These data confirm that the functional baroreflexes that normally maintain BP during orthostatic challenge are blocked by commonly used anesthetics.

特别声明

1、本页面内容包含部分的内容是基于公开信息的合理引用;引用内容仅为补充信息,不代表本站立场。

2、若认为本页面引用内容涉及侵权,请及时与本站联系,我们将第一时间处理。

3、其他媒体/个人如需使用本页面原创内容,需注明“来源:[生知库]”并获得授权;使用引用内容的,需自行联系原作者获得许可。

4、投稿及合作请联系:info@biocloudy.com。