Self-Trapped Excitons in Carbon Quantum Dots with Large NIR-II Photo-Thermoelectric Catalysis Induce Pyroptosis for Cancer Therapy

碳量子点中自陷激子与大近红外二区光热电催化作用诱导细胞焦亡用于癌症治疗

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Abstract

Pyroptosis, an immunogenic cell death mechanism triggered by Gasdermin family proteins, represents a transformative frontier in tumor immunotherapy. While carbon quantum dots (CQDs) have emerged as pyroptosis-triggering agents, their efficacy in NIR-II light-mediated therapy remains constrained by a low absorption coefficient and uncontrolled charge recombination. Herein, CQDs with high-density self-trapped excitons (STEs) exhibiting NIR-II absorption and an exceptional photothermal conversion efficiency of 51.2% are engineered. By optimizing the migration dynamics of hot carriers, directional charge separation is achieved, which generates cytotoxic hydroxyl radicals and superoxide radicals. The synergistic photo-thermoelectric catalysis triggers pyroptosis via reactive oxygen species-caspase 1-gasdermin D activation, eliciting robust systemic immunity that effectively eliminates the primary tumor and prevents tumor recurrence. This work establishes STE engineering as a universal design principle for advanced nanomaterials while pioneering a NIR-II-responsive pyroptosis platform that bridges localized ablation with systemic antitumor immunity, offering a paradigm shift for precision immuno-oncology.

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