Lipidomics and machine learning revealing dysregulation of specific triacylglycerol and phosphatidylglycerol as hub lipids associated with fetal growth in gestational diabetes mellitus

脂质组学和机器学习揭示了妊娠期糖尿病中与胎儿生长相关的特定三酰甘油和磷脂酰甘油的失调

阅读:1

Abstract

OBJECTIVE: Emerging evidence links lipid metabolism to the pathogenesis of gestational diabetes mellitus (GDM). This study aimed to identify lipidomic biomarkers and explore their clinical significance for GDM and related fetal growth and development through serum lipid profiling. METHODS: Lipidomic profiles of pregnant women with and without GDM were analyzed using Orthogonal Partial Least Squares Discriminant Analysis (OPLS-DA), Uniform Manifold Approximation and Projection (UMAP), volcano plots, and heatmaps. Carbon chain length and unsaturation effects on fold change (FC) were evaluated. Pathway analysis was performed via the Lipid Ontology (LION) platform, while lipid networks were constructed using Debiased Sparse Partial Correlation (DSPC). Hub lipids were identified through topological analysis and visualized with UpsetR. A GDM detection model was developed using Boruta and LogitBoost algorithms, assessed by receiver operating characteristic (ROC) curve analysis, and interpreted via Local Interpretable Model-agnostic Explanations (LIME). RESULTS: Twelve serum lipid metabolites were significantly associated with GDM risk. Phosphatidylglycerol (PG)(O-27:1) and triacylglycerol (TG)(35.5) were identified as hub lipids. The GDM detection model, incorporating TG(35:5), PG(O-27:1), total protein (TP), and red blood cell distribution width (RDW), achieved high accuracy. CONCLUSION: This study preliminarily characterized lipid metabolic pathway disturbances in patients with GDM, highlighting the potential of integrating lipidomics with interpretable machine learning techniques for biomarker discovery and mechanistic insight.

特别声明

1、本页面内容包含部分的内容是基于公开信息的合理引用;引用内容仅为补充信息,不代表本站立场。

2、若认为本页面引用内容涉及侵权,请及时与本站联系,我们将第一时间处理。

3、其他媒体/个人如需使用本页面原创内容,需注明“来源:[生知库]”并获得授权;使用引用内容的,需自行联系原作者获得许可。

4、投稿及合作请联系:info@biocloudy.com。