Association Between Chitinase 3-Like Protein 1 and Severe Nonalcoholic Fatty Liver Disease: A Large Prospective Cohort Study in UK Biobank

几丁质酶3样蛋白1与重度非酒精性脂肪肝疾病的关联:英国生物银行的一项大型前瞻性队列研究

阅读:3

Abstract

INTRODUCTION: Chitinase-3-like protein 1 (CHI3L1) is a glycoprotein involved in inflammation and fibrosis, but its association with nonalcoholic fatty liver disease (NAFLD) remains unclear. This study investigated the association between serum CHI3L1 levels and the risk of severe NAFLD in a large prospective cohort. METHODS: A prospective cohort study was conducted using UK Biobank data from 50,334 participants. Serum CHI3L1 levels were measured at baseline. Severe NAFLD cases were identified using hospital records. Cox proportional hazards models evaluated the association between CHI3L1 levels and severe NAFLD risk. Restricted cubic spline analysis assessed potential nonlinearity. Subgroup and mediation analyses were conducted to explore effect modifiers and underlying pathways. RESULTS: Over a median follow-up of 16 years, 766 severe NAFLD cases were identified. Higher CHI3L1 levels were significantly associated with increased severe NAFLD risk (hazard ratio 1.45, 95% confidence interval 1.34-1.58, P < 0.001). Restricted cubic spline analysis revealed a linear positive association without evidence of nonlinearity. Stratified analyses showed consistent associations across subgroups, with no significant interactions. Mediation analysis identified high-density lipoprotein cholesterol and alanine aminotransferase as partial mediators, explaining 6.38% and 2.86% of the total effect, respectively, whereas the direct effect of CHI3L1 remained dominant. DISCUSSION: Elevated CHI3L1 levels are associated with an increased risk of severe NAFLD. These findings suggest that CHI3L1 may serve as a novel biomarker and potential contributor to NAFLD progression, offering insights into the inflammatory and metabolic mechanisms underlying the disease.

特别声明

1、本页面内容包含部分的内容是基于公开信息的合理引用;引用内容仅为补充信息,不代表本站立场。

2、若认为本页面引用内容涉及侵权,请及时与本站联系,我们将第一时间处理。

3、其他媒体/个人如需使用本页面原创内容,需注明“来源:[生知库]”并获得授权;使用引用内容的,需自行联系原作者获得许可。

4、投稿及合作请联系:info@biocloudy.com。