Exercise-induced modulation of IGF-1 in healthy, obese, and cancer populations: a systematic review and meta-analysis

运动对健康人群、肥胖人群和癌症患者IGF-1的调节作用:系统评价和荟萃分析

阅读:1

Abstract

BACKGROUND: This systematic review and meta-analysis evaluated the effects of chronic exercise on circulating insulin-like growth factor 1 (IGF-1) across different populations, including, healthy adults, individuals with obesity, and cancer patients or survivors. To minimize confounding, we excluded trials combining exercise with medications, hormone therapy, or structured dietary interventions. MATERIALS AND METHODS: PubMed and Embase were searched through July 2024 for randomized controlled trials (RCTs) in adults (≥18 years) with exercise interventions lasting ≥8 weeks, a non-exercise control group, and reported changes in serum IGF-1. Twenty-one RCTs with 1376 participants met the inclusion criteria. Pooled weighted mean differences (WMD) with 95% confidence intervals (CI) were calculated, and trial sequential analysis was used to assess robustness. RESULTS: Exercise significantly increased IGF-1 in healthy individuals (WMD=21.41, 95% CI 8.01-34.81) and in those with obesity (WMD=15.46, 95% CI -1.07-31.99), consistent with metabolic and anabolic benefits via the GH-IGF-1 axis. In contrast, exercise significantly reduced IGF-1 in cancer patients or survivors (WMD=-14.71, 95% CI -19.77 to -9.65). In studies reporting both IGF-1 and IGF-binding protein 3 (IGFBP-3), exercise increased IGFBP-3 in healthy and cancer populations, suggesting a modulatory role of IGFBP-3 in IGF-1 regulation, particularly in cancer. CONCLUSION: Chronic exercise exerts health status-dependent effects on circulating IGF-1, supporting metabolic benefits in healthy and obese individuals and potentially contributing to cancer care by reducing IGF-1 in cancer patients or survivors. These findings demonstrate the complex endocrine response to exercise and support the therapeutic potential of tailored exercise prescriptions. .

特别声明

1、本页面内容包含部分的内容是基于公开信息的合理引用;引用内容仅为补充信息,不代表本站立场。

2、若认为本页面引用内容涉及侵权,请及时与本站联系,我们将第一时间处理。

3、其他媒体/个人如需使用本页面原创内容,需注明“来源:[生知库]”并获得授权;使用引用内容的,需自行联系原作者获得许可。

4、投稿及合作请联系:info@biocloudy.com。