Mutating heme oxygenase-1 into a peroxidase causes a defect in bilirubin synthesis associated with microcytic anemia and severe hyperinflammation

将血红素加氧酶-1突变为过氧化物酶会导致胆红素合成缺陷，并伴有小细胞性贫血和严重的炎症反应。