Regulating effect of genistein on the association of endothelial activation and stress index with atherosclerotic cardiovascular disease risk: a cross-sectional study of the NHANES database

染料木素对内皮激活和应激指数与动脉粥样硬化性心血管疾病风险关联的调节作用:一项基于NHANES数据库的横断面研究

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Abstract

The aim of this study is to assess the associations of urinary genistein and endothelial activation and stress index (EASIX) with 10-year atherosclerotic cardiovascular disease (ASCVD) risk. This cross-sectional analysis employed data obtained from the National Health and Nutrition Examination Survey (NHANES). Weighted univariable and multivariable logistic regression models examined EASIX-ASCVD risk correlations, with odds ratio and 95% confidence interval [OR (95%CI)] as effect measures. The potential interactions were explored between EASIX and genistein on 10-year ASCVD risk using additive and multiplicative interaction analyses. The relationships of EASIX and 10-year ASCVD risk were further assessed at different genistein levels. Subgroup analyses examined age, BMI, diabetes, hypertension, and dyslipidemia categories. Totally, 4478 eligible participants were stratified into low-risk (n = 2157) and high-risk (n = 2321) ASCVD groups. Elevated levels of EASIX (OR = 1.35, 95% CI 1.18-1.54), EASIX(median) ≥ 0.427 (OR = 1.40, 95% CI 1.14-1.73), EASIX(RCS) ≥ 0.434 (OR = 1.45, 95% CI 1.18-1.77), EASIX(quartile) (0.325-0.427, OR = 1.48, 95% CI 1.05-2.07; 0.427-0.567, OR = 1.51, 95% CI 1.08-2.10; ≥ 0.567, OR = 2.08, 95% CI 1.57-2.77), and log2(EASIX) (OR = 1.32, 95% CI 1.19-1.46) were associated with higher odds of 10-year ASCVD. Low genistein levels presented no significant correlation with 10-year ASCVD risk. The multiplicative interaction between EASIX and genistein levels was significant. When genistein levels < 54.105 µg/g, the connections between elevated EASIX levels and higher 10-year ASCVD risk were established in subgroups of age < 60 years, BMI ≥ 25 kg/m(2), non-diabetes, hypertension, and dyslipidemia. Elevated EASIX levels correlated with a higher 10-year ASCVD risk. Whether genistein modulates this association requires further validation, which may have potential implications for ASCVD prevention strategies in the general population.

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