Repression of PUM1-mediated mRNA decay activates translesion synthesis after DNA damage

PUM1介导的mRNA降解受到抑制,可在DNA损伤后激活跨损伤合成。

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Abstract

Biological roles of Pumilio1 (PUM1) in ubiquitous cells remain unclear. Here we identify 48 degrading target mRNAs by combined analysis of transcriptome-wide mRNA stabilities and the binding of mRNAs. Further analysis revealed that cells respond to DNA damage by inhibiting PUM1-mediated mRNA decay to activate translesion synthesis (46/50).

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