Timothy John Crow, OBE, PhD, DPM, FRCP, FRCPsych, FMedSci

蒂莫西·约翰·克劳,OBE,博士,足病医学博士,皇家内科医师学会会员,皇家精神科医师学会会员,英国医学科学院院士

阅读:1

Abstract

PURPOSE: Schizophrenia (SCZ) is a profound psychosomatic illness with an unidentified cause and no definitive biomarkers. This study sought to investigate plasma biomarkers linked to schizophrenia by untargeted metabolomics. PATIENTS AND METHODS: A total of 50 medication-naïve SCZ patients and 25 healthy controls were eligible and participated in this study. Psychiatric symptomatology was evaluated employing the Positive and Negative Syndrome Scale. We quantified the concentration of lipid metabolites in plasma from all participants using untargeted metabolomics and classified metabolites that were significantly different between both groups. We subsequently assessed the diagnostic potential of metabolites on the basis of receiver operating characteristic curves and examined metabolites affiliated with psychotic symptomatology in SCZ patients. RESULTS: Fourteen metabolites exhibited compelling disparities in schizophrenia patients relative to healthy controls. Eight metabolites, including methylphosphatidylcholine, acylcarnitine, sphingomyelin, and (O-acyl)-1-hydroxy fatty acids (38:4), could significantly distinguish between schizophrenia and healthy controls, with all their areas under the curve (AUC) exceeding 0.7. The peak area under the curve (CV-AUC) for AcCa(20:4) was 0.92 ± 0.03. In schizophrenia patients, negative symptoms exhibited a negative correlation with acylcarnitines, while cognitive symptoms had a substantial positive correlation with methylphosphatidylcholine and phosphatidylcholine. CONCLUSION: The findings reveal lipid metabolism dysregulation as a potential pathophysiological mechanism of schizophrenia. The identified metabolites, such as AcCa and phosphatidylcholine, serve as promising biomarkers for the diagnosis and symptom evaluation, suggesting their direct involvement in the disease's pathogenesis.

特别声明

1、本页面内容包含部分的内容是基于公开信息的合理引用;引用内容仅为补充信息,不代表本站立场。

2、若认为本页面引用内容涉及侵权,请及时与本站联系,我们将第一时间处理。

3、其他媒体/个人如需使用本页面原创内容,需注明“来源:[生知库]”并获得授权;使用引用内容的,需自行联系原作者获得许可。

4、投稿及合作请联系:info@biocloudy.com。