Probing the Role of Molecular Orientation on Spin-Selective Electron Transport through the Bacterial Multiheme Cytochrome MtrA

探究分子取向对细菌多血红素细胞色素MtrA自旋选择性电子传递的影响

阅读:1

Abstract

Extracellular electron transfer allows microbes to gain energy by transferring electrons between the cellular interior and external minerals that serve as electron acceptors or donors for respiration. Multiheme cytochromes, including the periplasmic decaheme MtrA, are key molecular conduits that mediate this biotic-abiotic electron exchange. Previous studies of electron transport through multiheme cytochromes adsorbed on ferromagnetic substrates demonstrated that these molecules exhibit the chirality induced spin selectivity (CISS) effect. This property is linked to efficient electron transfer by enhancing the transmission probability of one preferred electron spin, dependent on the chirality of the molecule. However, key questions remain about the factors that control the magnitude and sign of spin polarization in these molecules. While the effects of molecular size and secondary structure were previously analyzed, the roles of the molecule-substrate interface and orientation of the cytochromes were not investigated. Here, using conductive probe atomic force measurements of MtrA monolayers adsorbed onto ferromagnetic substrates, we demonstrate that both orientations of molecular adsorption on the magnetic substrate show higher conduction for the up-magnetization direction, indicating that the protein's preferred spin direction is independent of its orientation in the junction, unlike contrasting results previously reported for smaller chiral molecules. The maintained spin preference for opposite molecular orientations on the substrate indicates that the spin selectivity is controlled primarily by the inherent cytochrome chirality and structure rather than the precise coupling to the substrate.

特别声明

1、本页面内容包含部分的内容是基于公开信息的合理引用;引用内容仅为补充信息,不代表本站立场。

2、若认为本页面引用内容涉及侵权,请及时与本站联系,我们将第一时间处理。

3、其他媒体/个人如需使用本页面原创内容,需注明“来源:[生知库]”并获得授权;使用引用内容的,需自行联系原作者获得许可。

4、投稿及合作请联系:info@biocloudy.com。