EP12.10 HEMIZYGOUS DELETION OF CDKN2A/B IN IDH-MUTATED GLIOMA LEADS TO WORSE PROGNOSIS

EP12.10 IDH突变型胶质瘤中CDKN2A/B的半合子缺失导致预后更差

阅读:2

Abstract

BACKGROUND: The negative prognostic impact of CDKN2A/B homozygous deletion in IDH-mutated glioma is well established. However, the prognostic significance of a hemizygous deletion remains debated. This study aims to evaluate overall survival (OS) and clinical characteristics of IDH-mutated gliomas with a hemizygous deletion of CKDN2A/B, compared to patients with retained status. MATERIAL AND METHODS: This retrospective study defined the following inclusion criteria; (i) Operated at Sahlgrenska university hospital between 2007-2023 (ii) 18 years or older (iii) diagnosed with either an IDH-mutated astrocytoma or oligodendroglioma grade 2-3 according to the 2021 WHO classification system. CDKN2A/B status was determined through visual assessment of copy-number plots generated from DNA methylation data. The level of the CDKN2A/B locus was compared to baseline, whole chromosomal losses and extensive segmental losses to determine gene status. RESULTS: A total of 198 patients were included; 97 astrocytomas and 101 oligodendrogliomas. A hemizygous deletion of CDKN2A/B was identified in 21 cases. These patients had a significantly shorter OS compared to those with retained CDKN2A/B (8 years vs. 14 years, p = 0.02). This difference was significant in astrocytomas (n = 12, 6 years vs. 11 years, p = 0.03) but not in oligodendrogliomas (n = 9, 10 years vs. not reached, p = 0.27). Tumors with a hemizygous CDKN2A/B deletion presented more frequently with headache and motor deficits. However, in a sensitivity analysis, the distribution of debut symptom was only significant in oligodendrogliomas. There were no other significant differences regarding tumor volume, surgical approach or oncological treatment. CONCLUSION: A CDKN2A/B hemizygous deletion is associated with worse survival in IDH-mutated astrocytomas.

特别声明

1、本页面内容包含部分的内容是基于公开信息的合理引用;引用内容仅为补充信息,不代表本站立场。

2、若认为本页面引用内容涉及侵权,请及时与本站联系,我们将第一时间处理。

3、其他媒体/个人如需使用本页面原创内容,需注明“来源:[生知库]”并获得授权;使用引用内容的,需自行联系原作者获得许可。

4、投稿及合作请联系:info@biocloudy.com。