Salmonella modulates B cell biology to evade CD8(+) T cell-mediated immune responses

沙门氏菌通过调节B细胞生物学来逃避CD8(+) T细胞介导的免疫反应

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Abstract

Although B cells and antibodies are the central effectors of humoral immunity, B cells can also produce and secrete cytokines and present antigen to helper T cells. The uptake of antigen is mainly mediated by endocytosis; thus, antigens are often presented by MHC-II molecules. However, it is unclear if B cells can present these same antigens via MHC-I molecules. Recently, Salmonella bacteria were found to infect B cells, allowing possible antigen cross-processing that could generate bacterial peptides for antigen presentation via MHC-I molecules. Here, we will discuss available knowledge regarding Salmonella antigen presentation by infected B cell MHC-I molecules and subsequent inhibitory effects on CD8(+) T cells for bacterial evasion of cell-mediated immunity.

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