Mechanism-guided control of intracellular alkalinization and process pH for production of recombinant human B-type natriuretic peptide under the cSAT scheme in Escherichia coli

在大肠杆菌中,通过cSAT方案,以机制导向的方式控制细胞内碱化和工艺pH值,以生产重组人B型利钠肽。

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Abstract

BACKGROUND: Tag-based high-load expression in Escherichia coli can be accompanied by substantial drift of broth and intracellular pH, but mechanistic links between intracellular alkalinization, metabolism and process levers remain unclear. RESULTS: Using a cleavable self-aggregating tag (cSAT) to produce recombinant human B-type natriuretic peptide (rhBNP), we combine broth pH tracking, AF-C ratiometric intracellular pH imaging and multi-omics, which together suggest that ammoniagenic amino-acid catabolism, glyoxylate-centred carbon rerouting and respiratory shifts are major contributors to intracellular alkalinization. Guided by this proton-economy model, stepwise medium engineering (complex nitrogen, phosphate, glucose and ammonium sulfate with a tuned carbon to nitrogen ratio) lowers shake-flask broth pH from values above 8.5 to about 7.0, increases titers 2.9-fold over LB medium to 115.4 mg/L and, in 3-L fed-batch, yields 662.1 mg/L rhBNP while maintaining biomass. CONCLUSIONS: Proton-economy-based process design stabilizes pH and productivity during production of rhBNP under the cSAT scheme in E. coli. SUPPLEMENTARY INFORMATION: The online version contains supplementary material available at 10.1186/s12934-026-02938-7.

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