Strontium-loaded multifunctional gelatin methacryloyl hydrogels for type-H vascularized bone regeneration under osteoporotic conditions

用于骨质疏松条件下H型血管化骨再生的锶负载多功能明胶甲基丙烯酰水凝胶

阅读:1

Abstract

Osteoporosis (OP) is a clinically prevalent bone disease, under which excessive activation of osteoclasts (OCs) significantly delays bone regeneration. In this context, bone regeneration necessitates not merely the facilitation of osteogenesis and the suppression of resorption, but also more stringent requirements pertaining to vascularization, particularly type-H blood vessels. Recently, strontium (Sr) has emerged as a "dual-action bone agent", enhancing bone formation while suppressing bone resorption, yet still receives inadequate attention. Given that PDGF-BB secreted by osteoclast progenitors (pOCs) could induce type-H-related vascularization during coupling with osteogenesis, the regulatory effect of Sr(2+) on osteoclastogenesis need to be further studied and utilized in vascularized bone regeneration. Here, we synthesized Sr-substituted layered double hydroxide (Sr-LDH) by partially substituting divalent metal ions in LDH with Sr(2+), and encapsulated the surface-modified Sr-LDH into QK peptide-incorporated gelatin methacryloyl (GelMA) to form a composite hydrogel GelMA-QK/Sr-LDH@PDA (GLQ). This multifunctional hydrogel integrated the osteogenic and antiresorptive properties of Sr-LDH, exhibited significant bone regeneration efficacy under osteoporotic conditions. Furthermore, our study found that GLQ could stimulate bone marrow derived macrophages (BMMs) proliferation and preserve pOCs while suppress OC maturation, which further facilitated the expression of PDGF-BB and promoted type-H vessels development in the bone defect area. Taken together, the multifunctional composite hydrogel holds significant clinical implications for future treatment of osteoporotic bone defects.

特别声明

1、本页面内容包含部分的内容是基于公开信息的合理引用;引用内容仅为补充信息,不代表本站立场。

2、若认为本页面引用内容涉及侵权,请及时与本站联系,我们将第一时间处理。

3、其他媒体/个人如需使用本页面原创内容,需注明“来源:[生知库]”并获得授权;使用引用内容的,需自行联系原作者获得许可。

4、投稿及合作请联系:info@biocloudy.com。