Peripheral Blood Skin-Homing T Cells Define Immunological Endotypes in Atopic Dermatitis: A Case-Control Study in a Mexican Population

外周血皮肤归巢T细胞定义了特应性皮炎的免疫学内型:一项墨西哥人群病例对照研究

阅读:3

Abstract

BACKGROUND AND AIMS: Atopic dermatitis (AD) is a chronic inflammatory skin disease with marked immunological heterogeneity. T-cell subsets expressing CLA, CCR4, and CCR10 are involved in skin inflammation, but their frequency and expression patterns in AD remain poorly characterized. This study aimed to define distinct immunological endotypes of AD in the Mexican population by analyzing the frequency of CLA⁺ T cells expressing CCR4 and CCR10, along with their cytokine production profiles. METHODS: Flow cytometry quantified CD4⁺ and CD8⁺ T cells in 47 Mexican AD patients, 15 healthy controls, and 16 allergic individuals. Principal component analysis and unsupervised clustering were used to identify immunological patterns. RESULTS: Two AD endotypes were identified. Endotype 1 exhibited a higher frequency of CD4⁺CLA⁺ T cells (32.1% vs. 21.0%; p = 0.025) and a Th2/Th17-skewed profile, while Endotype 2 showed higher CD8⁺CLA⁺ T-cell frequencies (2.8% vs. 5.5%; p = 0.003) and increased chemokine receptor expression on CD4⁺ T cells (all p < 0.001). Clinically, Endotype 1 presented more active disease at examination (78.9% vs. 35.7%; p < 0.001). Total serum IgE was higher in Endotype 2 (5974 vs. 866 IU/mL; p = 0.032), and both endotypes showed higher IgE than healthy controls. SCORAD, POEM, and DLQI did not clearly separate the endotypes. CONCLUSION: AD presents distinct immunological endotypes characterized by specific T-cell profiles, which may inform personalized treatment strategies. This study provides insights into the immunological heterogeneity of AD in the Mexican population and underscores the need for population-specific approaches in disease characterization and management.

特别声明

1、本页面内容包含部分的内容是基于公开信息的合理引用;引用内容仅为补充信息,不代表本站立场。

2、若认为本页面引用内容涉及侵权,请及时与本站联系,我们将第一时间处理。

3、其他媒体/个人如需使用本页面原创内容,需注明“来源:[生知库]”并获得授权;使用引用内容的,需自行联系原作者获得许可。

4、投稿及合作请联系:info@biocloudy.com。