Nonequivalent response to bromodomain-targeting BET inhibitors in oligodendrocyte cell fate decision

少突胶质细胞命运决定中对溴结构域靶向BET抑制剂的反应不尽相同

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Abstract

The tandem bromodomains (BD1 and BD2) of the BET family proteins BRD2, BRD3, BRD4, and BRDT are structurally conserved but not functionally equivalent. In this issue of Chemistry & Biology, Gacias and colleagues report that a BD1-specific chemical inhibitor, Olinone, enhances oligodendrocyte differentiation, contrasting the reverse process triggered by broad BD1/BD2-targeting inhibitors, highlighting distinct roles of BD1 and BD2 in cell fate decision.

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