Mean platelet volume as a biomarker of anemia and platelet disorders among metabolic syndrome patients in Northwest Ethiopia

平均血小板体积作为埃塞俄比亚西北部代谢综合征患者贫血和血小板疾病的生物标志物

阅读:1

Abstract

Metabolic syndrome (MetS) is associated with increased cardiovascular risk, and platelets play a significant role in its pathophysiology. Mean platelet volume (MPV) is a marker of platelet activity. This study investigates the relationship between MPV and hematological abnormalities in MetS patients in Northwest Ethiopia. A cross-sectional study was conducted on adult MetS patients at the University of Gondar Comprehensive Specialized Hospital. Data on hematological parameters, anthropometric measurements, and biochemical profiles were collected using standard procedures. Six milliliters of venous blood were taken to conduct hematological and biochemical analyses, using an automated hematology and chemistry analyzer, respectively. Spearman's rank correlation and Mann-Whitney U tests were used to analyze associations between MPV and other variables. Statistical significance was determined at P-value < 0.05. Weak positive correlations were found between MPV and RBC count (ρ = 0.1588, P = 0.0018), Hgb level (ρ = 0.1621, P = 0.0014), and Hct (ρ = 0.2119, P < 0.0001), while a moderate negative correlation was observed between MPV and platelet count (ρ = -0.3988, P < 0.0001). MetS patients with anemia exhibited significantly lower median MPV values, while those with thrombocytopenia showed significantly higher median MPV values. Conversely, patients with thrombocytosis had significantly lower median MPV values compared to those without thrombocytosis. MPV is associated with hematological abnormalities in MetS patients in Northwest Ethiopia. Lower MPV was found in anemic MetS patients, while higher MPV was linked to thrombocytopenia, suggesting MPV's potential as a marker for assessing platelet dysfunction in MetS.

特别声明

1、本页面内容包含部分的内容是基于公开信息的合理引用;引用内容仅为补充信息,不代表本站立场。

2、若认为本页面引用内容涉及侵权,请及时与本站联系,我们将第一时间处理。

3、其他媒体/个人如需使用本页面原创内容,需注明“来源:[生知库]”并获得授权;使用引用内容的,需自行联系原作者获得许可。

4、投稿及合作请联系:info@biocloudy.com。