Associations of select neuropathologies with cardiovascular risk factors in a diverse cohort with autopsy‐confirmed Alzheimer disease

在经尸检确诊为阿尔茨海默病的多元化队列中,特定神经病理学与心血管危险因素的关联

阅读:1

Abstract

BACKGROUND: Cardiovascular risk factors have been implicated in the progression of Alzheimer disease (AD) and its neuropathological hallmarks, yet their specific contributions to regional brain pathology in diverse populations remain understudied. METHOD: In this cohort study (n = 276), we examined the relationship between three‐level categorizations (absent, active/recent, inactive/remote) of diabetes, hypertension, and hypercholesterolemia, and neuropathologies in a diverse group of Hispanic and non‐Hispanic White decedents with pathologically confirmed Intermediate/High AD from three Alzheimer's Disease Research Centers. Semi‐quantitative assessments for regional arteriolosclerosis, cerebral amyloid angiopathy (CAA) density, core plaques (CPs), diffuse plaques (DPs), and neuropil threads (NTs) were done adapting scales from established CERAD criteria. RESULT: Active diabetes was associated with increased density of frontal NTs (p <0.01), while active hypercholesterolemia correlated with greater density of CAA in temporal cortex (p = 0.04) and posterior hippocampus (p = 0.03), as well as temporal CPs (p = 0.03), as determined by Kruskal‐Wallis test. No significant associations were observed between hypertension status and neuropathology. Ordinal logistic regression adjusting for cardiovascular risk factors, ethnicity, sex, age of death, and center confirmed these associations except for CPs in the temporal cortex. CONCLUSION: This study underscores the importance of incorporating diverse cohorts in AD research to ensure broader generalizability and shows links between cardiovascular risk factors and AD‐related neuropathologies, advancing precision medicine approaches for dementia.

特别声明

1、本页面内容包含部分的内容是基于公开信息的合理引用;引用内容仅为补充信息,不代表本站立场。

2、若认为本页面引用内容涉及侵权,请及时与本站联系,我们将第一时间处理。

3、其他媒体/个人如需使用本页面原创内容,需注明“来源:[生知库]”并获得授权;使用引用内容的,需自行联系原作者获得许可。

4、投稿及合作请联系:info@biocloudy.com。