High-Fat Diet Exacerbates Neuroinflammation and Disease Severity in a Rat Model of Multiple Sclerosis

高脂肪饮食会加剧多发性硬化症大鼠模型中的神经炎症和疾病严重程度

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Abstract

BACKGROUND: Multiple sclerosis (MS) is an autoimmune inflammatory disorder of the central nervous system. The use of high-fat diet is a modifiable risk factor for a variety of inflammatory diseases. Thus, the aim of this study was to investigate the effects of a high-fat diet on the progression of MS in a validated animal model. METHODS: Adult Wistar rats (N = 10/group) were randomly divided into four groups: (1) standard diet, (2) high-fat diet, (3) and (4) standard and high-fat diet for 45 days, followed by induction of experimental encephalomyelitis. Animals were evaluated for clinical symptoms of MS and biochemical tests, including total cholesterol (TC), triglyceride (TG), low- and high-density lipoprotein-cholesterol (LDL-C and HDL-C), fibrinogen, interferon-gamma (IFN-γ), and interleukin-17 (IL-17). Tissue samples were subjected to neuropathological evaluation. The analysis was performed by one-way analysis of variance (ANOVA). RESULTS: The high-fat diet increased the progression of experimental autoimmune encephalomyelitis (EAE) disease, INF-γ (approximately 1.5 folds), IL-17 levels (approximately 3 folds), lymphocyte proliferation, TG (approximately 40 folds), TC (approximately 80 folds), LDL-C (approximately 14 folds), and fibrinogen levels (approximately 100 folds), but HDL-C (approximately 40 folds) was decreased (P < 0.05) in the affected MS group compared to standard diet. CONCLUSIONS: This study demonstrates that a high-fat diet significantly worsens clinical and immunological outcomes in an animal model of MS. The elevation of pro-inflammatory cytokines, particularly IFN-γ and IL-17, suggests that dietary fat may promote an inflammatory environment that accelerates disease progression. These findings highlight the need for further research to elucidate the underlying mechanisms.

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