Transcriptional coordination between intergenic RNA polymerase II-bound regions and nearby genes reveals functional specialization and disease associations in peripheral blood

基因间RNA聚合酶II结合区与邻近基因之间的转录协调揭示了外周血中的功能特化和疾病关联

阅读:1

Abstract

Recent studies have shown that RNA polymerase II (RNAPII) frequently occupies regions outside conventional promoters and gene bodies, forming intergenic RNAPII-bound regions (iRNAPII-BRs) that are actively transcribed and involved in gene regulation. We assessed the transcriptional activity of 181,547 iRNAPII-BRs from a genome-wide atlas and their impact on the expression of nearby gene in peripheral blood. The iRNAPII-BRs were often associated with structurally complex longer genes located in gene deserts. Their strongest transcriptional activity peaks occurred within 10 kb upstream from transcription start sites (TSSs), indicating a close regulatory link. Genes were grouped into six categories according to their transcriptional coordination with the nearest iRNAPII-BRs. Housekeeping genes tended to be transcribed independently, whereas blood tissue-specific genes were highly coordinated with iRNAPII-BR transcription. Genes with neuronal functions were frequently transcribed without active iRNAPII-BRs, suggesting basal promoter-driven expression with potential regulation by iRNAPII-BRs in response to blood-brain cross-talk cues. We found that 4507 iRNAPII-BRs were positively correlated with gene expression, whereas 793 were negatively correlated with gene expression implying a possible role in repression. The presence of transcription factor binding sites common to iRNAPII-BRs and promoters suggests a cooperative regulatory mechanism. Finally, we observed a differential expression of iRNAPII-BRs and nearby genes-ADRB2, CXCL8, SFN, and GPR3-in major depressive disorder, indicating that iRNAPII-BR-associated transcription may contribute to the pathophysiology of this disorder.

特别声明

1、本页面内容包含部分的内容是基于公开信息的合理引用;引用内容仅为补充信息,不代表本站立场。

2、若认为本页面引用内容涉及侵权,请及时与本站联系,我们将第一时间处理。

3、其他媒体/个人如需使用本页面原创内容,需注明“来源:[生知库]”并获得授权;使用引用内容的,需自行联系原作者获得许可。

4、投稿及合作请联系:info@biocloudy.com。