SERPINH1, a regulatory factor of oligodendroglioma cell invasion and extracellular matrix secretion, as a novel prognostic biomarker and therapeutic target

SERPINH1是少突胶质细胞瘤细胞侵袭和细胞外基质分泌的调控因子,可作为新型预后生物标志物和治疗靶点。

阅读:1

Abstract

BACKGROUND: Oligodendroglioma is a specific type of brain glioma, and relatively little research has been conducted on it. The expression and prognosis of serine protease peptidase inhibitor, branch H, member 1 (SERPINH1) in some malignant tumors have been studied, but the prognosis value and potential mechanism of this gene in oligodendroglioma have not been reported yet, and further research is needed. This study aims to reveal the expression characteristics and biological functions of SERPINH1 in oligodendroglioma cells, laying the foundation for targeted drug development. METHODS: Retrospective RNA sequencing (RNA-seq) data analysis was conducted in a cohort of 171 patients with oligodendroglioma in the Chinese Glioma Genome Atlas (CGGA) database and 149 patients in The Cancer Genome Atlas (TCGA) database. In addition, we used Gene Ontology (GO), Kyoto Encyclopedia of Genes and Genomes (KEGG), and Kaplan-Meier analyses, univariate and multivariate Cox regression analyses, correlation analysis, as well as Kolmogorov-Smirnov test and Unpaired t-test. RESULTS: SERPINH1 is highly enriched in short-lived patients compared to long-lived oligodendroglioma patients. SERPINH1 is relatively highly expressed in more malignant oligodendrogliomas. Functional enrichment shows that SERPINH1 is closely related to the tumor invasion function of oligodendroglioma. Further research has confirmed that SERPINH1 promotes the invasive function of tumor cells by regulating the secretion of extracellular matrix. Prognostic analysis shows that SERPINH1 is an independent poor prognostic factor for oligodendroglioma. CONCLUSIONS: We found that SERPINH1 can serve as an independent prognostic biomarker for oligodendroglioma. It may be a potential new target for the treatment of oligodendroglioma.

特别声明

1、本页面内容包含部分的内容是基于公开信息的合理引用;引用内容仅为补充信息,不代表本站立场。

2、若认为本页面引用内容涉及侵权,请及时与本站联系,我们将第一时间处理。

3、其他媒体/个人如需使用本页面原创内容,需注明“来源:[生知库]”并获得授权;使用引用内容的,需自行联系原作者获得许可。

4、投稿及合作请联系:info@biocloudy.com。