Potential role of (68)Ga- and (177)Lu-cyclic pentapeptides for in-vivo targeting CXCR4 receptor expression in chemotherapy relapse MCL patient

(68)Ga-和(177)Lu-环状五肽在化疗复发性套细胞淋巴瘤患者体内靶向CXCR4受体表达的潜在作用

阅读:1

Abstract

OBJECTIVES: High levels of CXCR4 expression in patients with mantle cell lymphoma is associated with poor prognosis. Various molecular techniques used are unable to specify the metastatic disease burden. Cyclic pentapeptides act as CXCR4 antagonists hence are functional markers of in-vivo CXCR4 receptor expression. In this view, the theragnostic complex of radiolabeled (68)Ga- and (177)Lu-cyclic pentapeptides was developed to in-vivo target the CXCR4 receptor expression. METHODS: Bone marrow aspiration and flow cytometry were performed to examine the fraction of lymphoid cells and immunophenotyping respectively. In-vitro CXCR4 receptor expression in the biopsied sample was determined using immunohistochemistry and flow cytometry molecular techniques. Diagnostic imaging using (68)Ga-cyclic pentapeptide was performed to check the in-vivo CXCR4 expression in chemotherapy relapse MCL patient. Dosimetry studies in the same patient was performed with different time-point imaging to calculate the residence time and predict the critical organ. RESULTS: Bone marrow aspiration indicated ~75% atypical lymphoid cells. Flow cytometric immunophenotyping revealed positivity for CD19, CD20, CD79b, Anti-kappa markers. IHC results showed high nuclear positivity. Approximately 86.11% of the cell population showed CXCR4 positive expression. Diagnostic imaging using (68)Ga-cyclic pentapeptide showed high tracer avidity in the mesenteric mass at L4 level. The avidity of both (68)Ga- and (177)Lu- cyclic pentapeptide radiotracers was noted in the mesenteric mass at the L4 level. Dosimetry study using (177)Lu-cyclic pentapeptide indicated kidneys as the critical organ with max residence time of 5.39 h. CONCLUSION: Theragnostic complex of radiolabelled (68)Ga/(177)Lu- cyclic pentapeptides have the potential to in-vivo target the CXCR4 receptor expression.

特别声明

1、本页面内容包含部分的内容是基于公开信息的合理引用;引用内容仅为补充信息,不代表本站立场。

2、若认为本页面引用内容涉及侵权,请及时与本站联系,我们将第一时间处理。

3、其他媒体/个人如需使用本页面原创内容,需注明“来源:[生知库]”并获得授权;使用引用内容的,需自行联系原作者获得许可。

4、投稿及合作请联系:info@biocloudy.com。