Astaxanthin-rich Nannochloropsis oculata mitigates high-fat diet-induced liver dysfunction in zebrafish by modulating lipid metabolism and oxidative stress

富含虾青素的眼状微拟球藻(Nannochloropsis oculata)通过调节脂质代谢和氧化应激,减轻高脂饮食引起的斑马鱼肝功能障碍。

阅读:1

Abstract

High-fat diets (HFDs) are widely used in aquaculture but may adversely affect fish liver health by disturbing lipid metabolism and promoting oxidative stress. This study investigated the protective effects of Nannochloropsis oculata enriched with astaxanthin on zebrafish (Danio rerio) fed an HFD. All zebrafish (n = 135) were divided randomly into three groups: conventional diet (CD), high-fat diet (HFD), and HFD supplemented with 2% N. oculata (HFD + NO). After a four-week feeding trial, growth performance, hepatosomatic index, liver oxidative stress markers, and expression of immune cytokine genes were evaluated. HFD group exhibited significantly increased hepatic lipid accumulation, elevated serum triglycerides and total cholesterol, and increased liver marker enzymes. These changes were accompanied by reduced antioxidant enzyme and non-enzymatic antioxidant activity, heightened oxidative stress, and upregulated pro-inflammatory cytokine expression (il-1β and il-8). Histological analysis confirmed hepatic steatosis and structural deterioration. Conversely, N. oculata supplementation significantly alleviated these effects by reducing hepatic lipid deposition, restoring antioxidant defenses, and downregulating pro-inflammatory cytokines while upregulating anti-inflammatory responses (IL-10 and TGF-β). These protective effects are attributed to astaxanthin's role in boosting mitochondrial β-oxidation, suppressing lipogenesis, and modulating inflammatory pathways. These findings suggest that N. oculata is a promising functional feed additive for mitigating diet-induced liver dysfunctions in aquaculture. Further research is needed to elucidate its molecular mechanisms and assess its long-term benefits across diverse aquatic environments.

特别声明

1、本页面内容包含部分的内容是基于公开信息的合理引用;引用内容仅为补充信息,不代表本站立场。

2、若认为本页面引用内容涉及侵权,请及时与本站联系,我们将第一时间处理。

3、其他媒体/个人如需使用本页面原创内容,需注明“来源:[生知库]”并获得授权;使用引用内容的,需自行联系原作者获得许可。

4、投稿及合作请联系:info@biocloudy.com。