Developmental and toxicological effects of butylated hydroxytoluene metabolites on zebrafish larvae

丁基羟基甲苯代谢物对斑马鱼幼体的发育和毒理学影响

阅读:1

Abstract

Butylated hydroxytoluene (BHT) is among the most widely used synthetic phenolic antioxidants. However, BHT and its metabolites have been detected in aquatic ecosystems, posing potential risks to aquatic organisms. The present study aimed to investigate the effects of BHT metabolites on embryonic development in zebrafish. To this end, embryos were exposed to BHT metabolites, including 3,5-di-tert-butyl-4 hydroxybenzaldehyde (BHT-CHO), 2,6-di-tert-butyl-4-(hydroxymethyl) phenol (BHT-OH), 3,5-di-tert-butyl-4 hydroxybenzoic acid (BHT-COOH), 2,6-di-tert-butyl-P-benzoquinone (BHT-Q), and 2,6-di-tert-butyl-4-hydroxy-4-methylcyclohexa-2,5-dien-1-one (BHT-quinol), from 1-120 h post-fertilization (hpf). BHT-CHO, -OH, -COOH, -Q, and -quinol were toxic to zebrafish larvae with 96 h LC(50) values of > 0.10, 15.85, 4.51, > 1.30, and 3.46 mg/L, respectively. Moreover, the acute toxicity of BHT metabolites to zebrafish larvae was indicated by morphological abnormalities, changes in heart rate, and alterations in locomotory behavior. The results indicated that exposure to BHT-COOH and BHT-OH caused intestinal developmental abnormalities, blood coagulation, tail deformities, and pericardial edema. Exposure to BHT-Q and BHT-quinol resulted in abnormal swim bladder development. Moreover, alterations in heart rate and locomotory behavior were observed in zebrafish larvae exposed to BHT-COOH, BHT-OH, and BHT-quinol. These findings demonstrate that exposure to BHT metabolites significantly affects the early growth and developmental stages of zebrafish larvae.

特别声明

1、本页面内容包含部分的内容是基于公开信息的合理引用;引用内容仅为补充信息,不代表本站立场。

2、若认为本页面引用内容涉及侵权,请及时与本站联系,我们将第一时间处理。

3、其他媒体/个人如需使用本页面原创内容,需注明“来源:[生知库]”并获得授权;使用引用内容的,需自行联系原作者获得许可。

4、投稿及合作请联系:info@biocloudy.com。