Synergistic changes in bystander CD8 and conventional CD4 T cells during neoadjuvant chemoimmunotherapy for non-small cell lung cancer reveal treatment response

非小细胞肺癌新辅助化疗免疫治疗期间旁观者 CD8 和常规 CD4 T 细胞的协同变化揭示了治疗反应

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Abstract

OBJECTIVE: We analyzed changes in intratumoral CD8(+) and CD4(+) T-cell subpopulations following neoadjuvant chemoimmunotherapy in non-small cell lung cancer. We then assessed whether these alterations favored better outcomes and explored their association with the tumor microenvironment. METHODS: Paired pre- and post-treatment samples from 32 patients with non-small cell lung cancer who underwent neoadjuvant chemoimmunotherapy at Shandong Cancer Hospital (January 2021-June 2023) were analyzed retrospectively. A quantitative analysis of tumor cells and their microenvironment was performed using a tissue microarray and a multiplex immunofluorescence technique. The analysis was based on the number of cells per thousand nucleated cells. Patients exhibiting a major pathologic response were classified as responders. The delta parameter (post-treatment minus pre-treatment) was utilized to assess changes in these indicators, and associations with treatment response were identified using the Wilcoxon Signed-Rank test and logistic regression analyses. RESULTS: Of the 32 patients, 59.38% were classified as responders. Across all patients, neoadjuvant chemoimmunotherapy significantly reduced the densities of dysfunctional CD8(+) resident memory T cells and cytotoxic and dysfunctional CD8(+) bystander T cells, while conventional CD4(+) T cells increased significantly. Similar trends were observed in the response group. In the non-response group, only cytotoxic CD8(+) bystander T cells were reduced in number. Logistic regression analysis revealed that a high delta conventional CD4(+) T cells is more favorable for MPR (OR = 0.13, p = 0.038), exhibiting a similar trend to changes in HIF-1α (p = 0.049). CONCLUSION: Alterations in specific CD8(+) and CD4(+) T-cell subpopulations during neoadjuvant chemoimmunotherapy may favor better outcomes and are potentially associated with tumor hypoxia. These findings provide a new perspective on developing strategies to improve treatment sensitivity in non-small cell lung cancer.

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