α-Synuclein deficiency promotes neuroinflammation by increasing Th1 cell-mediated immune responses
α-突触核蛋白缺乏通过增加 Th1 细胞介导的免疫反应来促进神经炎症
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作者：Benjamin Ettle, Kristina Kuhbandner, Stefanie Jörg, Alana Hoffmann, Jürgen Winkler, Ralf A Linker
Background
Increased α-synuclein immunoreactivity has been associated with inflammatory activity in multiple sclerosis (MS) lesions, but the function of α-synuclein in neuroinflammation remains unknown. The
Conclusions
Our findings indicate a functional role of α-synuclein in early EAE by increasing Th1 cell-mediated immune response.
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