Daratumumab and isatuximab differentially affect CD38 detection on plasma cells in myeloma: Anti-CD38 nanobody (clone JK36) and CD319 combination improve flow cytometric identification of plasma cells after targeted therapies

Daratumumab 和 isatuximab 对骨髓瘤浆细胞上 CD38 的检测具有不同的影响:抗 CD38 纳米抗体(克隆 JK36)和 CD319 联合使用可改善靶向治疗后浆细胞的流式细胞术鉴定。

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Abstract

Inclusion of daratumumab and isatuximab in therapeutic regimens has improved outcomes in multiple myeloma patients. As these agents block CD38 detection, they may result in difficulties in flow cytometric (FC) identification of plasma cells (PCs). In addition, the heterogeneity of myeloma immunophenotype and the introduction of novel therapeutic agents necessitate FC panels with robust gating markers. To this end, we validated an 11-colour PC panel that in addition to commonly used gating markers (CD45, CD38, CD138) includes CD319 and anti-CD38 single variable heavy domain nanobody (clone JK36). A Boolean "or" function was applied to gate PCs using a combination of the above markers. Patterns and intensity of antigen expression were compared in samples from patients receiving daratumumab or isatuximab therapy, or neither. Daratumumab resulted in loss of detectable CD38 expression (clone HB-7) on abnormal PCs, and JK36 overcame daratumumab blockage in almost all these cases. Interestingly, isatuximab showed an opposite pattern, with complete loss of CD38 detection by JK36, while retaining dim positivity by HB-7. This combination of markers proved highly effective in identifying abnormal PCs after CD38-directed therapies, supporting a need for redundancy of gating markers in FC panels.

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