Structural Alterations Associated With Cardiovascular Autonomic Failure in Multiple System Atrophy

多系统萎缩症中与心血管自主神经功能衰竭相关的结构改变

阅读:1

Abstract

BACKGROUND: Early severe autonomic failure (AF) in multiple system atrophy (MSA) is a risk factor for poor survival. Postmortem studies suggested that AF is related to the degeneration of preganglionic autonomic neurons of the brainstem and spinal cord. OBJECTIVES: Characterize cerebral alterations on brain imaging associated with cardiovascular AF. METHODS: Cardiovascular sympathetic failure was evaluated through orthostatic hypotension (OH) based on changes in systolic and diastolic blood pressure during tilt-test (ΔSBP and ΔDBP). Reduced heart rate (HR) variability reflecting cardio-vagal impairment was assessed with a composite score formed by the root-mean square differences of successive R-R intervals (RMSSD) and HR changes during deep breathing. Voxel-based morphometry (SPM12), volumetry, and cortical thickness measurements (FreeSurfer 7.0) of T1-weighted anatomical images were used to assess gray matter (GM) atrophy in sub-tentorial structures. Multivariate analysis included age, disease severity (UMSARS), and total intracranial volume as confounding factors. RESULTS: A total of 62 MSA patients followed at the French Reference Center were retrospectively included, aged 67.3 ± 8.6 years, 69.4% MSA-P, disease duration 4.2 ± 2.1 years. Medulla atrophy was correlated to OH (p < 0.006). Decrease in GM volume in the left anterior cerebellum (lobule V) was correlated to ΔDBP (pFWEc = 0.017). GM loss in the left interposed nucleus was correlated to ΔSBP (p < 0.003), whereas atrophy of the right dentate was associated with decreased HR variability (p < 0.003). CONCLUSION: Medulla volume was strongly correlated with OH. Cerebellar degeneration was associated with the severity of cardiovascular AF.

特别声明

1、本页面内容包含部分的内容是基于公开信息的合理引用;引用内容仅为补充信息,不代表本站立场。

2、若认为本页面引用内容涉及侵权,请及时与本站联系,我们将第一时间处理。

3、其他媒体/个人如需使用本页面原创内容,需注明“来源:[生知库]”并获得授权;使用引用内容的,需自行联系原作者获得许可。

4、投稿及合作请联系:info@biocloudy.com。