Dietary supplement use and regression from prediabetes to normoglycemia: insights from a population-based cohort study

膳食补充剂的使用与糖尿病前期向血糖正常化的逆转:一项基于人群队列研究的启示

阅读:2

Abstract

AIM: This study aimed to investigate the association between dietary supplement use and regression from pre-diabetes (Pre-DM) to normal glucose regulation (NGR) in a well-characterized population-based cohort. METHODS: A total of 2,174 adults with Pre-DM (mean age 51.9 ± 14.2 y; 48.6% men) were enrolled from the Tehran Lipid and Glucose Study (2009-2011) and followed up to 2015-2018. Baseline dietary supplement use was assessed via structured interviewer-administered questionnaires and verified when possible with supplement containers, medication lists, or written records. Participants reporting any supplement, including vitamins and minerals, were classified as users. Cox proportional hazards models were used to estimate hazard ratios (HRs) and 95% confidence intervals (CIs) for regression to NGR. The overall mean fasting serum glucose (FSG) and 2-hour serum glucose (2 h-SG) levels over time were compared between supplement users and non-users using repeated measures analysis of variance (ANOVA). RESULTS: Over a median of 6 years of follow-up, 44.2% of participants regressed to NGR. Calcium supplementation was associated with a 25% lower probability of regression (HR = 0.75, 95% CI = 0.59-0.96). Iron supplementation was associated with a 45% higher likelihood of regression to NGR (HR = 1.45, 95% CI = 1.05-2.02). Participants who reported taking iron supplements had a significantly lower mean FSG compared with non-users (99.7 vs. 104 mg/dL, P = 0.003), while mean 2 h-SG concentrations were slightly lower in this group (133 vs. 139 mg/dL, P = 0.09). CONCLUSION: Use of iron supplement may increase, whereas use of Ca supplement may decrease the likelihood of regression from Pre-DM to NGR.

特别声明

1、本页面内容包含部分的内容是基于公开信息的合理引用;引用内容仅为补充信息,不代表本站立场。

2、若认为本页面引用内容涉及侵权,请及时与本站联系,我们将第一时间处理。

3、其他媒体/个人如需使用本页面原创内容,需注明“来源:[生知库]”并获得授权;使用引用内容的,需自行联系原作者获得许可。

4、投稿及合作请联系:info@biocloudy.com。