Pathological complete response and long-term survival by pembrolizumab based immunochemotherapy in epidermal growth factor receptor mutated non-small cell lung cancer post-tyrosine kinase inhibitor failure: a case report and tumor microenvironment analysis

帕博利珠单抗联合免疫化疗治疗表皮生长因子受体突变型非小细胞肺癌,在酪氨酸激酶抑制剂治疗失败后获得病理完全缓解和长期生存:病例报告及肿瘤微环境分析

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Abstract

BACKGROUND: Lung adenocarcinoma harboring epidermal growth factor receptor (EGFR) mutations remains a significant clinical challenge, particularly when patients experience progression following EGFR-tyrosine kinase inhibitor (TKI) therapy. This case report explores the efficacy of pembrolizumab-based immunochemotherapy (ICT) in achieving a pathological complete response (pCR) and prolonged survival in a patient with EGFR-mutated non-small cell lung cancer (NSCLC) after TKI failure. CASE DESCRIPTION: A 74-year-old female patient with stage IVa EGFR L858R-mutated lung adenocarcinoma, progressing under multiple lines of EGFR-TKI therapy, was treated with pembrolizumab in combination with pemetrexed and carboplatin. Tumor response was assessed using radiological imaging and positron emission tomography-computed tomography (PET-CT). Pathological evaluation was conducted post-surgery, and the tumor microenvironment (TME) was analyzed using multiplex immunofluorescence (mIF) staining. Following four cycles of pembrolizumab-based ICT, the patient exhibited a significant reduction in tumor burden and mediastinal lymph node involvement, as confirmed by PET-CT. Surgical resection revealed a pCR with no viable tumor cells existed. The microenvironment analysis for tumor samples obtained post-progression on targeted therapy demonstrated that more than 50% of tumor cells expressing programmed cell death 1 ligand 1 (PD-L1), accompanying with higher infiltration of programmed death receptor 1 (PD-1)(+) CD8(+) T cells and PD-L1(+) CD68(+) macrophages in the tumor area compared to the stromal area. As for resected samples, substantial infiltration of CD45(+) immune cells, CD8(+) T lymphocytes, CD68(+) macrophages and immature tertiary lymphoid structures (TLSs) were detected. CONCLUSIONS: This case report highlights the potential of pembrolizumab-based ICT to induce a pCR and achieve long-term survival in EGFR-mutated NSCLC patients post-TKI failure. The favorable TME observed supports the rationale for this therapeutic approach and warrants further investigation in prospective clinical trials.

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