Relationship Between α-Glucosidase Inhibitors Application and Bone Mineral Density in Chinese Patients with Type 2 Diabetes Mellitus: A Cross-Sectional Study

α-葡萄糖苷酶抑制剂应用与中国2型糖尿病患者骨密度关系的研究:一项横断面研究

阅读:2

Abstract

PURPOSE: To investigate the correlation between α-glycosidase inhibitors (AGIs) and bone mineral density (BMD) in patients with type 2 diabetes mellitus (T2DM). PATIENTS AND METHODS: A total of 251 patients with T2DM admitted to the Zhongshan Hospital, Fudan University (Xiamen Branch) from September 2018 to September 2020 were enrolled. Baseline information of patients was analyzed according to different BMD subgroups. Clinical characteristics and BMD were compared between AGIs Group (n = 58) and non-AGIs group (n = 193). Multiple linear regression model was used to examine the relationship between AGIs application and BMD. RESULTS: The lower BMD group showed the characteristics of older age, longer duration of diabetes, lower body mass index (BMI) and estradiol (E2). In the AGIs group, the proportion of females, duration of diabetes, incidence of diabetic peripheral vascular disease and the use of sulfonylureas were significantly higher than those in the non-AGIs group (p < 0.05), the results of HbA1c, TC, TG, LDL, FT3, FT4 were opposite (p < 0.05). Compared with the non-AGIs group, the BMD of femoral neck and lumbar spine in the AGIs group was significantly decreased, FRAX score and the prevalence of osteoporosis were remarkably increased, accompanied by a decrease in β-CTX and P1NP level. Multivariate linear regression analysis showed a significant negative correlation between AGIs and lumbar BMD after adjustment for potential confounding variables (β = -0.053, 95% CI -0.100~0.006, P = 0.029). CONCLUSION: This study indicates that the use of AGIs in patients with T2DM is significantly associated with an increased risk of BMD decline, osteoporosis, and fracture.

特别声明

1、本页面内容包含部分的内容是基于公开信息的合理引用;引用内容仅为补充信息,不代表本站立场。

2、若认为本页面引用内容涉及侵权,请及时与本站联系,我们将第一时间处理。

3、其他媒体/个人如需使用本页面原创内容,需注明“来源:[生知库]”并获得授权;使用引用内容的,需自行联系原作者获得许可。

4、投稿及合作请联系:info@biocloudy.com。