Long, saturated chains: tasty domains for kinases of insulin resistance

长链饱和脂肪酸:胰岛素抵抗激酶的理想靶点

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Abstract

The mechanistic basis of how cells respond to increased fatty acids (FAs) is murky but potentially involves receptor-mediated activation or inhibition by different FA classes. Holzer et al. (2011) recently propose in Cell that expansion of intracellular membrane microdomains induced by saturated FA recruit and activate c-Src for JNK activation.

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