Endoscopic decompression of L3 nerve root in a case of adult spinal deformity correction with post-operative neurological deficit: a case report

成人脊柱畸形矫正术后出现神经功能障碍:内镜下L3神经根减压术一例报告

阅读:1

Abstract

Adult spinal deformities (ASD) have a very high incidence of postoperative neurological decline. Spine surgeons all over the world are broadening the indications of endoscopic spine surgery (ESS) with similar/better outcomes to other conventional spine surgeries. In this report we present a case of L3-radiculopathy post ASD correction and its management with ESS. A 78-year-old female operated for ASD presented with backache, and right-side-dominant radiating leg pain. Imaging showed distal junctional failure with left rod fracture between L5-S1 and screw loosening with gross sagittal imbalance. She was treated with L4 pedicle subtraction osteotomy (PSO) with T10-iliac posterior fixation with use of satellite rods at PSO site. Post-operatively she developed left-sided L3 motor deficit. Imaging revealed left L3 root compression by bone fragment near the left L3 pedicle screw-head. We presume that, since the L3 pedicular screw had to be inserted deeper than the other screws to accommodate the satellite-rod, the bone in the area near the screw head (part of either the pedicle or facet) caused compression of the left L3 nerve root. The patient was treated with interlaminar ESS under awake anaesthesia and the compressing bone was drilled out. Post-operatively, patient had rapid relief of the radicular pain and improvement in the left quadriceps (L3) power with no recurrence at 1-year follow-up. Thus, post-operative iatrogenic neurological deficits can be treated successfully using ESS when the exact cause of the deficit is identified and is amenable to endoscopic removal. We could successfully treat a case of left-sided L3-radiculopathy, caused due to protruding bony spur after ASD correction surgery with ESS.

特别声明

1、本页面内容包含部分的内容是基于公开信息的合理引用;引用内容仅为补充信息,不代表本站立场。

2、若认为本页面引用内容涉及侵权,请及时与本站联系,我们将第一时间处理。

3、其他媒体/个人如需使用本页面原创内容,需注明“来源:[生知库]”并获得授权;使用引用内容的,需自行联系原作者获得许可。

4、投稿及合作请联系:info@biocloudy.com。