Fatty acids alter to the toxicity of islet amyloid polypeptide aggregates in a length and saturation dependent manner

脂肪酸以长度和饱和度依赖的方式改变胰岛淀粉样多肽聚集体的毒性。

阅读:2

Abstract

Abrupt the aggregation of islet amyloid polypeptide (IAPP), a 37 amino acid hormone, is an expected molecular cause of type 2 diabetes (T2D), a severe pathology that affects more than 30 million Americans. During the past decade, a significant increase in T2D was observed in children, teens, and young adults. Although this increase is associated with changes in nutrition and lifestyle, its origin remains unclear. Our results indicate that fatty acids often used as food supplements drastically accelerate IAPP aggregation and increase the cytotoxicity of IAPP aggregates. Using C. elegans that overexpress IAPP as a model system, we show that an increase in the amount of fatty acids in nematode diets drastically shortens their lifespan. These results indicate that fatty acids present in the diet can accelerate the onset and progression of T2D.

特别声明

1、本页面内容包含部分的内容是基于公开信息的合理引用;引用内容仅为补充信息,不代表本站立场。

2、若认为本页面引用内容涉及侵权,请及时与本站联系,我们将第一时间处理。

3、其他媒体/个人如需使用本页面原创内容,需注明“来源:[生知库]”并获得授权;使用引用内容的,需自行联系原作者获得许可。

4、投稿及合作请联系:info@biocloudy.com。