Molecular Characterization of Carbapenem and Colistin Resistance in Isolates Causing Urinary Tract Infections at an Outpatient Setting of a Tertiary Care Hospital

一家三级医院门诊尿路感染分离株中碳青霉烯类和粘菌素耐药性的分子特征分析

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Abstract

Background Increasing resistance to higher-end antibiotics like carbapenems and colistin is a great cause of concern. There is a paucity of data on the detection of carbapenem and colistin in outpatient settings. This study was to determine the prevalence of carbapenem-resistant Enterobacterales (CRE) and colistin resistance in CRE at the outpatient setting of a tertiary care institute of North India. Methods A prospective study was conducted from January to December 2020. A total of 6,829 non-repetitive urine samples were cultured on cysteine lactose electrolyte-deficient (CLED) agar. Identification was done by matrix-assisted laser desorption ionization-time of flight mass spectrometry (MALDI-TOF MS), and antimicrobial susceptibility testing followed Clinical and Laboratory Standards Institute (CLSI) 2020, using Kirby-Bauer and Vitek 2 (BioMérieux, Marcy-l'Étoile, France). Carbapenem-resistant isolates were screened for carbapenemase genes (bla(NDM) , bla(KPC) , bla(OXA) , bla(OXA-48) ). Colistin resistance was confirmed by broth microdilution. Results A total of 669/6829 (9.79%) urine specimens yielded significant growth, mostly in females (54.1%), less than in other studies probably due to fewer samples received because of the COVID-19 pandemic. The majority (50.82%) of patients belonged to the early adult group (15-45 years). Most organisms were Enterobacteriaceae (509/669, 76.08%). The most common organism isolated was E. coli (56.5%) followed by K. pneumoniae (18.2%). 5.97% (40/669) isolates were carbapenem-resistant. 6.87% (35/509) of Enterobacteriaceae were carbapenem-resistant, which is less than in other studies possibly due to samples from outpatient settings. The following carbapenemase genes were detected: bla(NDM)  (18/35), bla(KPC)  (2/35), bla(OXA ) (11/35) and bla(OXA-48)  (5/35). Some isolates had co-existence of more than two carbapenemase genes. Colistin resistance in CRE was 11% which is like other studies conducted in the same tertiary centre. Comparison of resistance patterns of other antibiotics in CRE and carbapenem-sensitive Enterobacterales (CSE) group shows that fosfomycin, ticarcillin-clavulanate and tetracycline had higher sensitivity in the CRE group. Conclusion The presence of these genes in outpatient settings is worrisome. Both bla (NDM )and bla(OXA-48) resulted in higher minimum inhibitory concentrations (MICs) against carbapenems. Co-presence of NDM with OXA-48-producing E. coli in urine culture. Colistin resistance in an outpatient setting is alarming. Early detection of these resistance-determinant genes by molecular methods is essential in limiting the spread of infection due to these organisms.

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